Effects of Beta Cypermethrin on Enzyme Activities of Zebrafish (Brachydanio Rerio)
- DOI
- 10.2991/emcm-16.2017.18How to use a DOI?
- Keywords
- Beta cypermethrin; Zebrafish; Mortality; SOD; POD
- Abstract
In this paper, zebrafish was used as a model organism to study the toxicity of beta cypermethrin with different concentration gradient on the whole zebrafish, and the effects on superoxide dismutase (SOD) and peroxidase (POD) activities of zebrafish brain tissue. As a result, was found among the mortality rate of zebrafish and the concentration or exposure time of beta cypermethrin, and thus showed a dose - effect or time - effect relationship between each other. The correlation coefficient of dose - effect relationship (>0.9) showed that the dose of beta cypermethrin was significantly related to its toxic effect on zebrafish. The 96 h LC50 of beta cypermethrin was 0.650 mg/L, and so beta cypermethrin was a highly toxic chemical according to the chemical toxicity classification standard of fish acute toxicity experiment. In addition, a linear negative correlation was found between the damage degree of tissues and the decrease extent of enzyme activities of zebrafish and the concentration and/or extension time of beta cypermethrin. In particular, beta cypermethrin had an inhibitory effect on SOD activity, and a suppressing effect after induction on POD activity.
- Copyright
- © 2017, the Authors. Published by Atlantis Press.
- Open Access
- This is an open access article distributed under the CC BY-NC license (http://creativecommons.org/licenses/by-nc/4.0/).
Cite this article
TY - CONF AU - Jia Jin AU - Songling Zhang AU - Pengpeng Wang AU - Min Zhang PY - 2017/02 DA - 2017/02 TI - Effects of Beta Cypermethrin on Enzyme Activities of Zebrafish (Brachydanio Rerio) BT - Proceedings of the 2016 7th International Conference on Education, Management, Computer and Medicine (EMCM 2016) PB - Atlantis Press SP - 86 EP - 92 SN - 2352-538X UR - https://doi.org/10.2991/emcm-16.2017.18 DO - 10.2991/emcm-16.2017.18 ID - Jin2017/02 ER -